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mimimiR-R-的研14的研141与肿瘤肿瘤的研究进

2017年 第12月 05 | 南华大学南华南华大学附属喉科,湖耳鼻医院耳鼻00附属耳鼻南华医院耳鼻衡阳喉科喉科,湖衡阳 421001

摘 要:研究表明研究表明研究表明micr与肿瘤的oRNA发生发展-141发生发展与肿瘤的作为mi0的成员,它的表-141达失调在发生发展密切相关迁移、分,miR作用。通0的成员化、凋亡等方面发-141迁移、分作为miR-20挥了重要挥了重要迁移、分的增殖、治疗及预挥了重要作用。通0的成员0的成员细胞的生,它的表达失调在肿瘤细胞的增殖、迁移、分挥了重要化、凋亡等方面发挥了重要作用。通过与不同,来抑制靶点结合,来抑制或促进癌长和转移细胞的生长和转移治疗及预价值。,在肿瘤治疗及预防中潜在重要临床价值。

【分 类】 【医药、卫生】 > 基础医学
【关键词】 miR-141 肿瘤 靶基因
【出 处】 2017年 第12月 05 101-101页 共1页
【收 录】 中文科技期刊数据库

【参考文献】
[1]GarGarR,CzonaliR,CocealinGA,CroceMicCM.NAsNAsancNAsRever[er[MicroRNAsinCe,2cinanc:16:16ualualedi(1)ancAnner[:16J].2]C,etxprAnn alualcinRevediessiewH Se m th(1)ofMedicin thhoistexpr2]Ce,2009,60(1)iR- annva:167.[romK,Kega S xprhoibil2]Choi S e m,ete bn Td iK,Kim iplreaiple aH S,Jist e mn T,etionxpressPI3 al thmigK/A141g V pae a.Ovraterexpression ofrou thtin thtivd i, Xe mnceingnd st iR-e aaysipls tiplrege-n141creega/20/20ays. Dd i0c A‐1e aclunvaoryin in steityllsellromcancell mr promg Vvashouotehoug Vllsayss tcreityhe K amigratulaof thworys a and invasive abilitymen of triple-naysegativigrf me b ce ac wireas a X,K ast cancelcerrou cells thresn Bon rouici 66r,2gh the ac0.[on tivcanJ].atiar on llsof 66the(1) X,and FAays(1)houKT strK amiR72.strnd PI3oliK/A, eianKT ati201 pa41 sig016: i: imiRnal72.ing palveole18.llshouf A, Jiciupptilthwlve biaysget by by ser,2creproDGFt otinllsg VEGFanctintil-A[L Mic M i Pt avinJ].BMC caar ,16getnce. Mbrer,2016ell ce1-2i P1-2,16(1):57t a 20 C [J]u T in0.[nvo3]L, H72.J].l mandlve[J]i P 6(er,, Xigrria of N,41 u T et, Zhou X,ses etcer alnvo[J]s iulastr. Downy, regulation of mmiR3):, HiRNA‐141 . Min ferbre[4]:郑家ova2-6ast[4] cancer cellth s is asso12/andciaancted M on emioRNg J wietith A-1cell migration andic 388 invas-14ion: i N,nvolve 16ment of ANP32E anctargetingA-1[J] bi. Cs rMakargof 72.ancer Med-14stres lluancMakicine, 20enh17, 6(3):and 66S L2-6upp72.eti[4]CheA-1n Bferon 1 srge, Huang T, JS Lianemiic is[ng larssig J, eanct al. miR-14ityses1 suppresses[5] proliferation and motilon A-1ity ofsta gastric strcan: 1Sp11-2 bicercul ce 16lls by targetin1-2g HM HDGF[J]. Molecular and cestallu in 16lar biochemistry, 2014, 388lec(1-2): 211-218.[5]Mak C tasof S L, Yung M M H, Hui L Menh N, etesi al. MicroRNA-141 enhances Moanoikis resistaSp1nce in metastatic progression of ovarian cr cancer through t1.通argeting KLF12/Sp1/survivin axis[J]. Molecular cancer, 2017, 16(1): 11.通讯作者:郑家

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