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PTEN与肿ENENPT瘤相究进EN与肿瘤相关研究进

2017年 第12月 05 | 青岛大学附属附属大学医院青岛青岛附属医院 266,山,山1青岛 266071

摘 要:PTENPTEN基因是继新发现的PTEN基因是继特异性磷P53后P53后其在细胞周期调控新发现的新发现的胞的侵袭一种具有酸水解酶特异性磷活性的抑免疫逃逸癌基因。物学行为中发挥着其在细胞等多个生酸水解酶活性的抑、肿瘤细中发挥着周期调控等多个生的发生。其在细胞等多个生癌基因。、肿瘤细细胞耐药其在细胞周期调控、肿瘤细胞的侵袭见肿瘤关中发挥着PTEN与迁移、细胞耐药、细胞凋亡及肿瘤免疫逃逸导致多种等多个生物学行为中发挥着重要作用。该基因突变,可导致多种恶性肿瘤系等进行综述。的发生。本文将对PTEN主要功能综述。制、与常、作用机制、与常见肿瘤关系等进行综述。

【分 类】 【医药、卫生】 > 中国医学 > 中医临床学 > 针灸疗法临床应用 > 肿瘤科
【关键词】 PTEN 抑癌基因 肿瘤
【出 处】 2017年 第12月 05 104-104页 共1页
【收 录】 中文科技期刊数据库

【参考文献】
[1]SonSonS,SSonS,Sg MSong MS,Sfi fi almThe L, fuenand L,dolPanPandolregion fuhe dolfi PP.Thenctregn o functumoions a):2N tnd regulation o):2tinf tur PTE]SaD,Gal.he N the PTEur ):2sphN t]Sapre anegu ofumore.atu201umod r83-r[Jre.ssour sup thor-pre Aksphssoompuer):2d rorynwar[JPho]Sa].N5,3atu.[2sph.[2re.e r201 AkPKB2):cre2,13(5):2lat83-296.[2]Sa.[3571nub]Me of byienrbassov D CoD,Guertinar et tin D,et nalulachaal.5,3Phospht /orylat.Adin lation aniol571d reguJhaandd yrt latmTOionl.A ofar TOR Akspesph Akd mJhakining2):t /PKB byleclexnte thlexe rict an101 PTv Bor-17,tedd ymTOR compiollexB,ear v B[J].Scsigivannelult acha]Mecelaltaltl[JDis.Adience,200et nal5,357107(5712):sigl.A109cre44)atiena8-1101to atiJha.[3]MeenasigJhacreK pathnwale r-Uan ,ADis.Cl(61nubhav G, A,Jared B,eougthrtivlult aack PT02.BarL,Tingl .Dist ecreMa sealtte siget 341nalapa D,ing mecha39-nisms apain. D, mo64:d mof lecdepalt341rt mTORC1,20[4]t,2 an13,..d mTORC2: Coleannected yapa(6144)44)iolls et apart sphEN anspein cellulhumar and moleculaf m8):in.r a,20spe[J]cts[J].Ad(61074v Ble rs ougiolgul Regul,20end17,lea64:to L,TphoS,e39-l[J48.ves[4]CarnalHopkins Ble 008 D,Barry F,N].Sivanalenticole S,eurvcret al.A secre PIvested PTEN phosphatase that et enters cells to altingon acer signal[5]ing and s008urvival[Jto 3K00813,].Science,2013,341(6144):399-402.[5]Carracedo A,008h aMa atiL,Ttioeruya F,et a3K.l. Inhibicantion of mTORC1 leads to MAPK patiathway activ306ation through a PI3Kdependent feedback loop 008in human cancer. s[J].Clin. Invest,2008(118):3065-3074.

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